INTRODUCTION:
Fahr’s disease or Fahr’s syndrome is characterized by the calcification
of basal ganglia and cerebral cortex, which is usually transmitted in an
autosomal dominant manner. It is a rare neurological condition and most
of the patients with the disease condition present with movement
disorders, tremors, dyskinesia, and sometimes gait disturbances,
cognitive impairment, speech disturbances, psychiatric changes such as
mania, sensory changes or even pain [1-3]. Fahr’s
disease should be differentiated from Fahr’s syndrome, which is usually
secondary to underlying systemic pathology, this could be differentiated
with blood tests for serum calcium, iPTH, along with other routine blood
investigations. Moreover, basal ganglia calcification seen in CT scans
could radiologically mimic the disease condition, thus clinical
correlation with radiological investigations are the key in the
diagnosis of Fahr’s disease [2, 4, 5]. Here, we
present a case of a 65-year-old male, with Fahr’s syndrome secondary to
hyperparathyroidism which initially mimicked as Parkinson’s disease and
later improved after starting the patient on vitamin D and calcium
carbonate therapy.